Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Then again, our capacity to rescue SERT operate in KI midbrain synaptoneurosomes with the inhibition of FAK suggests elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by elevated pFAK localization in 5-HT synapses. https://jessicar270isy6.wikievia.com/user